CONFERENCE PROCEEDING
Smoking history and long-term outcomes post PCI by sex, from FU-Registry
| 1 | School of Medicine, Fukuoka University, Kyushu, Japan |
CORRESPONDING AUTHOR
Publication date: 2019-10-12
Tob. Induc. Dis. 2019;17(Suppl 1):A43
KEYWORDS
ABSTRACT
Objective:
Most important lifestyle factor for the primary and secondary prevention for coronary heart diseases is smoking. However, few reported on the relationship between smoking habits/history and long-term outcomes post PCI procedures.
Methods:
From our PCI- Registry (FU-Registry), 829 PCI cases (497 males, 332 females) whose 5 years follow-up data including clinical outcomes were available, were used.
Results:
In males, no difference was observed in patient’s background or lesion characteristics between smoker and never smoker groups, however, smoking (+) showed high incidence of dyslipidemia, statin use at first PCI, but as for clinical outcomes are similar between the groups. For females, smoking (+) group showed low HDL-C (48.3 +12.8mg/dL vs. 51.5+13.4mg/dL, p<0.01), and lesion reference was significantly smaller than smoking (-) group. No difference was observed in medications, while smoking (+) showed high complication of ASO.
Conclusions:
Females were less smokers than males, while female smokers showed low HDL-C levels at PCI and then longterm outcomes (including MACEs) were more frequent in smokers than never smokers.
Most important lifestyle factor for the primary and secondary prevention for coronary heart diseases is smoking. However, few reported on the relationship between smoking habits/history and long-term outcomes post PCI procedures.
Methods:
From our PCI- Registry (FU-Registry), 829 PCI cases (497 males, 332 females) whose 5 years follow-up data including clinical outcomes were available, were used.
Results:
In males, no difference was observed in patient’s background or lesion characteristics between smoker and never smoker groups, however, smoking (+) showed high incidence of dyslipidemia, statin use at first PCI, but as for clinical outcomes are similar between the groups. For females, smoking (+) group showed low HDL-C (48.3 +12.8mg/dL vs. 51.5+13.4mg/dL, p<0.01), and lesion reference was significantly smaller than smoking (-) group. No difference was observed in medications, while smoking (+) showed high complication of ASO.
Conclusions:
Females were less smokers than males, while female smokers showed low HDL-C levels at PCI and then longterm outcomes (including MACEs) were more frequent in smokers than never smokers.
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